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在庫・価格 : 2025年05月26日 05時04分 現在

商品名 商品コード メーカー 包装 価格 在庫 リスト
Anti-Synaptopodin, Mouse-Mono(G1D4)
データシート
61094 POGプロジェン バイオテクニック
PROGEN Biotechnik GmbH
50 μg ¥91,000
(未発注)
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在庫・価格 : 2025年05月26日 05時04分 現在

Anti-Synaptopodin, Mouse-Mono(G1D4)

  • 商品コード:61094
  • メーカー:POG
  • 包装:50μg
  • 価格: ¥91,000
  • 在庫:無(未発注)
使用文献
No. 文献情報 備考 参照
1 Batchelder CA et al. Natural Scaffolds for Renal Differentiation of Human Embryonic Stem Cells for Kidney Tissue Engineering. PLoS ONE 2015;10(12):e0143849
Batchelder CA et al
2015/01/01
PubMed
2 Krall P et al. Podocyte-specific overexpression of wild type or mutant trpc6 in mice is sufficient to cause glomerular disease. PLoS ONE 2010;5(9):e12859
Krall P et al
2010/01/01
PubMed
3 Asanuma K et al. Synaptopodin regulates the actin-bundling activity of alpha-actinin in an isoform-specific manner. J. Clin. Invest. 2005 May;115(5):1188-98
Asanuma K et al
2005/01/01
PubMed
4 Kihara I et al. Origin of hyperplastic epithelial cells in idiopathic collapsing glomerulopathy. Histopathology 1999 Jun;34(6):537-47
Kihara I et al
1999/01/01
PubMed
5 Barisoni L et al. The dysregulated podocyte phenotype: a novel concept in the pathogenesis of collapsing idiopathic focal segmental glomerulosclerosis and HIV-associated nephropathy. J. Am. Soc. Nephrol. 1999 Jan;10(1):51-61
Barisoni L et al
1999/01/01
PubMed
6 Kobayashi N et al. Nonuniform microtubular polarity established by CHO1/MKLP1 motor protein is necessary for process formation of podocytes. J. Cell Biol. 1998 Dec;143(7):1961-70
Kobayashi N et al
1998/01/01
PubMed
7 Mundel P et al. Rearrangements of the cytoskeleton and cell contacts induce process formation during differentiation of conditionally immortalized mouse podocyte cell lines. Exp. Cell Res. 1997 Oct;236(1):248-58
Mundel P et al
1997/01/01
PubMed
8 Mundel P et al. Synaptopodin: an actin-associated protein in telencephalic dendrites and renal podocytes. J. Cell Biol. 1997 Oct;139(1):193-204
Mundel P et al
1997/01/01
PubMed
9 Mundel P et al. Induction of differentiation in cultured rat and human podocytes. J. Am. Soc. Nephrol. 1997 May;8(5):697-705
Mundel P et al
1997/01/01
PubMed
10 Mundel P et al. Structure and function of podocytes: an update. Anat. Embryol. 1995 Nov;192(5):385-97
Mundel P et al
1995/01/01
PubMed
11 Mundel P et al. Podocytes in glomerulus of rat kidney express a characteristic 44 KD protein. J. Histochem. Cytochem. 1991 Aug;39(8):1047-56
Mundel P et al
1991/01/01
PubMed
12 Matsusaka T et al. Angiotensin receptor blocker protection against podocyte-induced sclerosis is podocyte angiotensin II type 1 receptor-independent. Hypertension 2010 Apr;55(4):967-73
Matsusaka T et al
2010/01/01
PubMed
13 Wagner N et al. The podocyte protein nephrin is required for cardiac vessel formation. Hum. Mol. Genet. 2011 Jun;20(11):2182-94
Wagner N et al
2011/01/01
PubMed
14 Turk T et al. BMP signaling and podocyte markers are decreased in human diabetic nephropathy in association with CTGF overexpression. J. Histochem. Cytochem. 2009 Jul;57(7):623-31
Turk T et al
2009/01/01
PubMed
15 Schiwek D et al. Stable expression of nephrin and localization to cell-cell contacts in novel murine podocyte cell lines. Kidney Int. 2004 Jul;66(1):91-101
Schiwek D et al
2004/01/01
PubMed
16 Jain N et al. TLR-mediated albuminuria needs TNFα-mediated cooperativity between TLRs present in hematopoietic tissues and CD80 present on non-hematopoietic tissues in mice. Dis Model Mech 2016 Jun;9(6):707-17
Jain N et al
2016/01/01
PubMed
17 Wang L et al. Gq signaling causes glomerular injury by activating TRPC6. J. Clin. Invest. 2015 May;125(5):1913-26
Wang L et al
2015/01/01
PubMed
18 Tanaka E et al. Notch2 activation ameliorates nephrosis. Nat Commun 2014;5:3296
Tanaka E et al
2014/01/01
PubMed
19 Koop K et al. Selective loss of podoplanin protein expression accompanies proteinuria and precedes alterations in podocyte morphology in a spontaneous proteinuric rat model. Am. J. Pathol. 2008 Aug;173(2):315-26
Koop K et al
2008/01/01
PubMed
20 Wagner KD et al. An inducible mouse model for PAX2-dependent glomerular disease: insights into a complex pathogenesis. Curr. Biol. 2006 Apr;16(8):793-800
Wagner KD et al
2006/01/01
PubMed
21 Patek CE et al. Murine Denys-Drash syndrome: evidence of podocyte de-differentiation and systemic mediation of glomerulosclerosis. Hum. Mol. Genet. 2003 Sep;12(18):2379-94
Patek CE et al
2003/01/01
PubMed
22 Ihara K et al. MAGI-2 is critical for the formation and maintenance of the glomerular filtration barrier in mouse kidney. Am. J. Pathol. 2014 Oct;184(10):2699-708
Ihara K et al
2014/01/01
PubMed
23 Peitsch WK et al. Cell biological and biochemical characterization of drebrin complexes in mesangial cells and podocytes of renal glomeruli. J. Am. Soc. Nephrol. 2003 Jun;14(6):1452-63
Peitsch WK et al
2003/01/01
PubMed
24 Walter B et al. Differential expression pattern of protein ARVCF in nephron segments of human and mouse kidney. Histochem. Cell Biol. 2008 Nov;130(5):943-56
Walter B et al
2008/01/01
PubMed
25 Turk, T. et al. BMP Signaling and Podocyte Markers Are Decreased in Human Diabetic Nephropathy in Association With CTGF Overexpression. J. Histochem. Cytochem. 57, 623-631 (2009).

PubMed
26 Matsusaka, T. et al. ARB Protection Against Podocyte-Induced Sclerosis is Podocyte AT1-Independent. Hypertension 55, 967-973 (2010).

PubMed
27 Ihara, K. I. et al. MAGI-2 is critical for the formation and maintenance of the glomerular filtration barrier in mouse kidney. Am. J. Pathol. 184, 2699-2708 (2014).

PubMed
28 Toffoli, B. et al. Hemicentin 1 influences podocyte dynamic changes in glomerular diseases. Am.J.Physiol.Renal.Physiol. 314, F1154-F1165 (2018)

PubMed
29 Peitsch, W. K. Cell biological and biochemical characterization of drebrin complexes in mesangial cells and podocytes of renal glomeruli. J. Am. Soc. Nephrol. 14, 1452-1463 (2003).

PubMed
30 Krall, P. et al. Podocyte-Specific Overexpression of Wild Type or Mutant Trpc6 in Mice Is Sufficient to Cause Glomerular Disease. PLoS One 5, (2010).

PubMed
31 Zhu Z et al. Mitoquinone Protects Podocytes from Angiotensin II-Induced Mitochondrial Dysfunction and Injury via the Keap1-Nrf2 Signaling Pathway. Oxid Med Cell Longev 2021;2021:1394486
Zhu Z et al
2021/01/01
PubMed
32 Tsuchida, Y. et al. Lipoprotein modulation of proteinuric renal injury. Lab.Invest. , (2019)

PubMed
33 Held, M. et al. Ex vivo live cell tracking in kidney organoids using light sheet fluorescence microscopy. PLoS.One. 13, e0199918 (2018)

PubMed
34 Puliti, A. et al. Albuminuria and Glomerular Damage in Mice Lacking the Metabotropic Glutamate Receptor 1. Am. J. Pathol. 178, 1257-1269 (2011).

PubMed
35 Collino, F. et al. Preeclamptic sera induce nephrin shedding from podocytes through endothelin-1 release by endothelial glomerular cells. Am. J. Physiol. - Ren. Physiol. 294, 1185-1194 (2008).

PubMed
36 Gauer, S. et al. CTGF Is Expressed During Cystic Remodeling in the PKD/Mhm (cy/+) Rat Model for Autosomal-Dominant Polycystic Kidney Disease (ADPKD). J.Histochem.Cytochem. 65, 743-755 (2017)

PubMed
37 Patek, C. E. et al. Murine Denys-Drash syndrome: evidence of podocyte de-differentiation and systemic mediation of glomerulosclerosis. Hum. Mol. Genet. 12, 2379-2394 (2003).

PubMed
38 Ornellas, F. et al. Mesenchymal Stromal Cells Induce Podocyte Protection in the Puromycin Injury Model. Sci.Rep. 9, 19604 (2019)

PubMed
39 Xavier S et al. BAMBI is expressed in endothelial cells and is regulated by lysosomal/autolysosomal degradation. PLoS ONE 2010;5(9):e12995
Xavier S et al
2010/01/01
PubMed
40 Hirose T et al. An essential role of the universal polarity protein, aPKClambda, on the maintenance of podocyte slit diaphragms. PLoS One 2009;4(1):e4194
Hirose T et al
2009/01/01
PubMed
41 Meyer-Schwesinger, C. et al. Nephrotic Syndrome and Subepithelial Deposits in a Mouse Model of Immune-Mediated Anti-Podocyte Glomerulonephritis. J. Immunol. 187, 3218-3229 (2011).

PubMed
42 Wagner, K.-D. et al. An Inducible Mouse Model for PAX2-Dependent Glomerular Disease: Insights into a Complex Pathogenesis. Curr. Biol. 16, 793-800 (2006).

PubMed
43 Canales CP et al. Characterization of a Trpc6 Transgenic Mouse Associated with Early Onset FSGS. Br J Med Med Res 2015;5(10):1198-2012
Canales CP et al
2015/01/01
PubMed
44 Schell C et al. ARP3 Controls the Podocyte Architecture at the Kidney Filtration Barrier. Dev Cell 2018 Dec;47(6):741-757.e8
Schell C et al
2018/01/01
PubMed
45 Mundel, P., Gilbert, P. & Kriz2, W. Podocytes in Glomerulus of Rat Kidney Express a Characteristic 44 KD Protein-. J. Histochem. Cytochem. 39, 1047-1056 (1991).

PubMed
46 Mundel, P. et al. Rearrangements of the cytoskeleton and cell contacts induce process formation during differentiation of conditionally immortalized mouse podocyte cell lines. Exp. Cell Res. 236, 248-58 (1997).

PubMed
47 G旦del M et al. Role of mTOR in podocyte function and diabetic nephropathy in humans and mice. J. Clin. Invest. 2011 Jun;121(6):2197-209
G旦del M et al
2011/01/01
PubMed
48 Rachubik P et al. The TRPC6-AMPK Pathway is Involved in Insulin-Dependent Cytoskeleton Reorganization and Glucose Uptake in Cultured Rat Podocytes. Cell Physiol Biochem 2018;51(1):393-410
Rachubik P et al
2018/01/01
PubMed
49 Barisoni, L., Kriz, W., Mundel, P. & D‘Agati, V. The dysregulated podocyte phenotype: a novel concept in the pathogenesis of collapsing idiopathic focal segmental glomerulosclerosis and HIV-associated nephropathy. J. Am. Soc. Nephrol. 10, 51-61 (1999).

PubMed
50 Zhou, J. et al. Functional comparison of distinct Brachyury+ states in a renal differentiation assay. Biol.Open. 7,- (2018).

PubMed
51 Schiwek, D. et al. Stable expression of nephrin and localization to cell-cell contacts in novel murine podocyte cell lines. Kidney Int. 66, 91-101 (2004).

PubMed
52 Prochnicki, A. et al. Characterization of Glomerular Sox9+ Cells in Anti-Glomerular Basement Membrane Nephritis in the Rat. Am.J.Pathol. 188, 2529-2541 (2018)

PubMed
53 Manigrasso, M. et al. Deletion of the formin Diaph1 protects from structural and functional abnormalities in the murine diabetic kidney. Am.J.Physiol.Renal.Physiol. 315, F1601-F1612 (2018)

PubMed
54 Wagner, N. et al. The podocyte protein nephrin is required for cardiac vessel formation. Hum. Mol. Genet. 20, 2182-2194 (2011).

PubMed
55 Haley, K. et al. Podocyte injury elicits loss and recovery of cellular forces. Sci.Adv. 4, eaap8030 (2018).

PubMed
56 Kihara, I. et al. Origin of hyperplastic epithelial cells in idiopathic collapsing glomerulopathy. Histopathology 34, 537-47 (1999).

PubMed
57 Ekulu PM et al. Novel Human Podocyte Cell Model Carrying G2/G2 APOL1 High-Risk Genotype. Cells 2021 Jul;10(8)
Ekulu PM et al
2021/01/01
PubMed
58 Fukuda, A. et al. Urinary podocyte and TGF-?1 mRNA as markers for disease activity and progression in anti-glomerular basement membrane nephritis. Nephrol.Dial.Transplant. 32, 1818-1830 (2017).

PubMed
59 Endlich, N. et al. The transcription factor Dach1 is essential for podocyte function. J.Cell.Mol.Med. 22, 2656-2669 (2018).

PubMed
60 An, J. et al. Urinary myo-inositol is associated with the clinical outcome in focal segmental glomerulosclerosis. Sci.Rep. 9, 14707 (2019)

PubMed
61 Armelloni, S. et al. NeuroD Expression in Podocytes and Interrelationships with Nephrin at Both Nuclear and Cytoplasmic Sites. Cell.Physiol.Biochem. 46, 873-889 (2018)

PubMed
  • No.: 1
  • 文献情報:
    Batchelder CA et al. Natural Scaffolds for Renal Differentiation of Human Embryonic Stem Cells for Kidney Tissue Engineering. PLoS ONE 2015;10(12):e0143849
    Batchelder CA et al
    2015/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 2
  • 文献情報:
    Krall P et al. Podocyte-specific overexpression of wild type or mutant trpc6 in mice is sufficient to cause glomerular disease. PLoS ONE 2010;5(9):e12859
    Krall P et al
    2010/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 3
  • 文献情報:
    Asanuma K et al. Synaptopodin regulates the actin-bundling activity of alpha-actinin in an isoform-specific manner. J. Clin. Invest. 2005 May;115(5):1188-98
    Asanuma K et al
    2005/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 4
  • 文献情報:
    Kihara I et al. Origin of hyperplastic epithelial cells in idiopathic collapsing glomerulopathy. Histopathology 1999 Jun;34(6):537-47
    Kihara I et al
    1999/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 5
  • 文献情報:
    Barisoni L et al. The dysregulated podocyte phenotype: a novel concept in the pathogenesis of collapsing idiopathic focal segmental glomerulosclerosis and HIV-associated nephropathy. J. Am. Soc. Nephrol. 1999 Jan;10(1):51-61
    Barisoni L et al
    1999/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 6
  • 文献情報:
    Kobayashi N et al. Nonuniform microtubular polarity established by CHO1/MKLP1 motor protein is necessary for process formation of podocytes. J. Cell Biol. 1998 Dec;143(7):1961-70
    Kobayashi N et al
    1998/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 7
  • 文献情報:
    Mundel P et al. Rearrangements of the cytoskeleton and cell contacts induce process formation during differentiation of conditionally immortalized mouse podocyte cell lines. Exp. Cell Res. 1997 Oct;236(1):248-58
    Mundel P et al
    1997/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 8
  • 文献情報:
    Mundel P et al. Synaptopodin: an actin-associated protein in telencephalic dendrites and renal podocytes. J. Cell Biol. 1997 Oct;139(1):193-204
    Mundel P et al
    1997/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 9
  • 文献情報:
    Mundel P et al. Induction of differentiation in cultured rat and human podocytes. J. Am. Soc. Nephrol. 1997 May;8(5):697-705
    Mundel P et al
    1997/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 10
  • 文献情報:
    Mundel P et al. Structure and function of podocytes: an update. Anat. Embryol. 1995 Nov;192(5):385-97
    Mundel P et al
    1995/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 11
  • 文献情報:
    Mundel P et al. Podocytes in glomerulus of rat kidney express a characteristic 44 KD protein. J. Histochem. Cytochem. 1991 Aug;39(8):1047-56
    Mundel P et al
    1991/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 12
  • 文献情報:
    Matsusaka T et al. Angiotensin receptor blocker protection against podocyte-induced sclerosis is podocyte angiotensin II type 1 receptor-independent. Hypertension 2010 Apr;55(4):967-73
    Matsusaka T et al
    2010/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 13
  • 文献情報:
    Wagner N et al. The podocyte protein nephrin is required for cardiac vessel formation. Hum. Mol. Genet. 2011 Jun;20(11):2182-94
    Wagner N et al
    2011/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 14
  • 文献情報:
    Turk T et al. BMP signaling and podocyte markers are decreased in human diabetic nephropathy in association with CTGF overexpression. J. Histochem. Cytochem. 2009 Jul;57(7):623-31
    Turk T et al
    2009/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 15
  • 文献情報:
    Schiwek D et al. Stable expression of nephrin and localization to cell-cell contacts in novel murine podocyte cell lines. Kidney Int. 2004 Jul;66(1):91-101
    Schiwek D et al
    2004/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 16
  • 文献情報:
    Jain N et al. TLR-mediated albuminuria needs TNFα-mediated cooperativity between TLRs present in hematopoietic tissues and CD80 present on non-hematopoietic tissues in mice. Dis Model Mech 2016 Jun;9(6):707-17
    Jain N et al
    2016/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 17
  • 文献情報:
    Wang L et al. Gq signaling causes glomerular injury by activating TRPC6. J. Clin. Invest. 2015 May;125(5):1913-26
    Wang L et al
    2015/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 18
  • 文献情報:
    Tanaka E et al. Notch2 activation ameliorates nephrosis. Nat Commun 2014;5:3296
    Tanaka E et al
    2014/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 19
  • 文献情報:
    Koop K et al. Selective loss of podoplanin protein expression accompanies proteinuria and precedes alterations in podocyte morphology in a spontaneous proteinuric rat model. Am. J. Pathol. 2008 Aug;173(2):315-26
    Koop K et al
    2008/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 20
  • 文献情報:
    Wagner KD et al. An inducible mouse model for PAX2-dependent glomerular disease: insights into a complex pathogenesis. Curr. Biol. 2006 Apr;16(8):793-800
    Wagner KD et al
    2006/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 21
  • 文献情報:
    Patek CE et al. Murine Denys-Drash syndrome: evidence of podocyte de-differentiation and systemic mediation of glomerulosclerosis. Hum. Mol. Genet. 2003 Sep;12(18):2379-94
    Patek CE et al
    2003/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 22
  • 文献情報:
    Ihara K et al. MAGI-2 is critical for the formation and maintenance of the glomerular filtration barrier in mouse kidney. Am. J. Pathol. 2014 Oct;184(10):2699-708
    Ihara K et al
    2014/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 23
  • 文献情報:
    Peitsch WK et al. Cell biological and biochemical characterization of drebrin complexes in mesangial cells and podocytes of renal glomeruli. J. Am. Soc. Nephrol. 2003 Jun;14(6):1452-63
    Peitsch WK et al
    2003/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 24
  • 文献情報:
    Walter B et al. Differential expression pattern of protein ARVCF in nephron segments of human and mouse kidney. Histochem. Cell Biol. 2008 Nov;130(5):943-56
    Walter B et al
    2008/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 25
  • 文献情報:
    Turk, T. et al. BMP Signaling and Podocyte Markers Are Decreased in Human Diabetic Nephropathy in Association With CTGF Overexpression. J. Histochem. Cytochem. 57, 623-631 (2009).

  • 備考:
  • 参照:
    PubMed
  • No.: 26
  • 文献情報:
    Matsusaka, T. et al. ARB Protection Against Podocyte-Induced Sclerosis is Podocyte AT1-Independent. Hypertension 55, 967-973 (2010).

  • 備考:
  • 参照:
    PubMed
  • No.: 27
  • 文献情報:
    Ihara, K. I. et al. MAGI-2 is critical for the formation and maintenance of the glomerular filtration barrier in mouse kidney. Am. J. Pathol. 184, 2699-2708 (2014).

  • 備考:
  • 参照:
    PubMed
  • No.: 28
  • 文献情報:
    Toffoli, B. et al. Hemicentin 1 influences podocyte dynamic changes in glomerular diseases. Am.J.Physiol.Renal.Physiol. 314, F1154-F1165 (2018)

  • 備考:
  • 参照:
    PubMed
  • No.: 29
  • 文献情報:
    Peitsch, W. K. Cell biological and biochemical characterization of drebrin complexes in mesangial cells and podocytes of renal glomeruli. J. Am. Soc. Nephrol. 14, 1452-1463 (2003).

  • 備考:
  • 参照:
    PubMed
  • No.: 30
  • 文献情報:
    Krall, P. et al. Podocyte-Specific Overexpression of Wild Type or Mutant Trpc6 in Mice Is Sufficient to Cause Glomerular Disease. PLoS One 5, (2010).

  • 備考:
  • 参照:
    PubMed
  • No.: 31
  • 文献情報:
    Zhu Z et al. Mitoquinone Protects Podocytes from Angiotensin II-Induced Mitochondrial Dysfunction and Injury via the Keap1-Nrf2 Signaling Pathway. Oxid Med Cell Longev 2021;2021:1394486
    Zhu Z et al
    2021/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 32
  • 文献情報:
    Tsuchida, Y. et al. Lipoprotein modulation of proteinuric renal injury. Lab.Invest. , (2019)

  • 備考:
  • 参照:
    PubMed
  • No.: 33
  • 文献情報:
    Held, M. et al. Ex vivo live cell tracking in kidney organoids using light sheet fluorescence microscopy. PLoS.One. 13, e0199918 (2018)

  • 備考:
  • 参照:
    PubMed
  • No.: 34
  • 文献情報:
    Puliti, A. et al. Albuminuria and Glomerular Damage in Mice Lacking the Metabotropic Glutamate Receptor 1. Am. J. Pathol. 178, 1257-1269 (2011).

  • 備考:
  • 参照:
    PubMed
  • No.: 35
  • 文献情報:
    Collino, F. et al. Preeclamptic sera induce nephrin shedding from podocytes through endothelin-1 release by endothelial glomerular cells. Am. J. Physiol. - Ren. Physiol. 294, 1185-1194 (2008).

  • 備考:
  • 参照:
    PubMed
  • No.: 36
  • 文献情報:
    Gauer, S. et al. CTGF Is Expressed During Cystic Remodeling in the PKD/Mhm (cy/+) Rat Model for Autosomal-Dominant Polycystic Kidney Disease (ADPKD). J.Histochem.Cytochem. 65, 743-755 (2017)

  • 備考:
  • 参照:
    PubMed
  • No.: 37
  • 文献情報:
    Patek, C. E. et al. Murine Denys-Drash syndrome: evidence of podocyte de-differentiation and systemic mediation of glomerulosclerosis. Hum. Mol. Genet. 12, 2379-2394 (2003).

  • 備考:
  • 参照:
    PubMed
  • No.: 38
  • 文献情報:
    Ornellas, F. et al. Mesenchymal Stromal Cells Induce Podocyte Protection in the Puromycin Injury Model. Sci.Rep. 9, 19604 (2019)

  • 備考:
  • 参照:
    PubMed
  • No.: 39
  • 文献情報:
    Xavier S et al. BAMBI is expressed in endothelial cells and is regulated by lysosomal/autolysosomal degradation. PLoS ONE 2010;5(9):e12995
    Xavier S et al
    2010/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 40
  • 文献情報:
    Hirose T et al. An essential role of the universal polarity protein, aPKClambda, on the maintenance of podocyte slit diaphragms. PLoS One 2009;4(1):e4194
    Hirose T et al
    2009/01/01
  • 備考:
  • 参照:
    PubMed
  • No.: 41
  • 文献情報:
    Meyer-Schwesinger, C. et al. Nephrotic Syndrome and Subepithelial Deposits in a Mouse Model of Immune-Mediated Anti-Podocyte Glomerulonephritis. J. Immunol. 187, 3218-3229 (2011).

  • 備考:
  • 参照:
    PubMed
  • No.: 42
  • 文献情報:
    Wagner, K.-D. et al. An Inducible Mouse Model for PAX2-Dependent Glomerular Disease: Insights into a Complex Pathogenesis. Curr. Biol. 16, 793-800 (2006).

  • 備考:
  • 参照:
    PubMed
  • No.: 43
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